Vagal maneuvers: Someone with supraventricular tachycardia (abnormally rapid heart rate originating just above the ventricles) may be taught vagal maneuvers, which slow down the heartbeats and help the heart resume its normal electrical flow.Blood thinners lower the risk of clotting and stroke. Blood thinners: AFib, whether persistent or intermittent, should be treated with anticoagulant medications (blood thinners) based on the CHA 2DS 2-VASc score.The type and location of a reentry arrhythmia determine how it will be treated. These mutations affect the nature of electrical pathways in the heart, making them more likely to become circuits.Ī 2020 study suggests that mutations of genes such as SCN1A, PRKAG2, RYR2, CFTR, NOS1, PIK3CB, GAD2, and HIP1R may affect pathways that produce AVNRT. Similarly, scar tissue that forms after tissue is damaged during a heart attack can also become a reentry circuit.Ībnormalities involving electrolytes, such as sodium and potassium, can cause structural changes in the heart that become reentry circuits.Ĭertain gene mutations are also associated with reentry arrhythmias. Scar tissue that results from any type of heart surgery has the potential to become a new pathway for electrical impulses. Other individuals born with unrelated congenital heart defects may develop a reentry circuit following surgery to repair a different structural problem within the heart. Some people are born with an extra pathway, though the arrhythmia may not develop until later in life. Several conditions can create reentry circuits that then lead to reentry arrhythmias. What causes reentry arrhythmia and who’s at risk? The main cause of a reentry arrhythmia is the presence of an extra electrical pathway, called a reentrant circuit, that keeps impulses moving in a circle, re-exciting tissue instead of letting it rest in between contractions. Bundle branch ways then carry electrical impulses to stimulate the ventricles, causing them to pump blood out to your lungs and the rest of your body.Ī reentry arrhythmia occurs when at least some of the impulses constantly traveling through your heart’s electrical network don’t pause and move on as they should, but instead loop back around and stimulate the atria or ventricles again and again.Ī heart can be in a state of reentry arrhythmia for very brief or extended episodes. The impulses pause, and then spread out in electrical pathways known as the bundle of His. The electrical impulses also move down to the atrioventricular node located between the atria and ventricles. The sinus node causes the atria to contract and pump blood down to the lower chambers (ventricles). The sinus node is located near the top of the heart in the right atria. The normal pattern of electrical activity in your heart begins with the sinus node generating an electrical stimulus. This article reviews the different types of reentry arrhythmia, their symptoms, causes, treatment, and outlook. It’s often possible to treat a reentry arrhythmia with medications and procedures that block the circuit and restore healthy electrical activity in the heart. There are several different types of reentry arrhythmias, some of which can lead to severe cardiac complications. When a closed-loop circuit interrupts the normal flow of electrical impulses in the heart, troubling symptoms and health complications can follow. Arrhythmias can cause the heart to beat abnormally fast or slow or in a chaotic, irregular manner.Ī reentry arrhythmia is one in which electrical impulses stray from their usual path and repeatedly return to the tissue that generated them, essentially forming a closed-loop circuit. A disruption in that pattern is called an arrhythmia. The electrical impulses that keep your heart beating in a steady, consistent rhythm follow the same path throughout the heart dozens of times per minute. Treatment can usually resolve these types of arrhythmias.
A reentry arrhythmia is an abnormality in your heart’s rhythm in which electrical impulses in your heart stray from their usual path and form a closed-loop circuit.
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